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来源：百度知道 编辑：UC知道 时间：2024/06/30 09:30:57
Colonic secretion of oxalate is stimulated in rat models exhibiting hyperoxalemia suggesting a contribution of this extrarenal pathway to regulation of oxalate mass balance in clinical conditions manifesting hyperoxalemia. The transport avenues and regulatory mechanisms may not be identical to those observed during adaptive enteric oxalate secretion in chronic renal failure models.
Hyperoxaluria(高草酸尿) alone (CH) was not associated with changes in colonic oxalate(草酸盐) transport, whereas changes in net oxalate(草酸盐 transport in distal colon(末端结肠) from absorption to net secretion was observed in models with hyperoxalemia (CHH and AH). Angiotensin II receptor (血管紧张素Ⅱ受体）antagonism（拮抗作用) with losartan(氯沙坦) rereduced net colonic oxalate secretion in AH but not CHH.
Hyperoxaluria(高草酸尿) alone (CH) was not associated with changes in colonic oxalate(草酸盐) transport, whereas changes in net oxalate(草酸盐 transport in distal colon(末端结肠) from absorption to net secretion was observed in models with hyperoxalemia (CHH and AH). Angiotensin II receptor (血管紧张素Ⅱ受体）antagonism（拮抗作用) with losartan(氯沙坦) rereduced net colonic oxalate secretion in AH but not CHH.
草酸盐的 Colonic 分泌在鼠模型中被刺激展现在临床的情况建议一个这一 extrarenal 的贡献通往草酸盐块平衡的规则的道路显示 hyperoxalemia 的 hyperoxalemia 。 运输系统大街和管制的机制在慢性的肾脏失败模型中不可能和被在适合的肠草酸盐分泌期间观察的那些相同。
乙二酸酯的大肠分泌物在陈列hyperoxalemia的鼠模型在体现hyperoxalemia的临床情况被刺激建议这条extrarenal路的贡献到乙二酸酯配重的章程.运输大道和管理机制在慢性肾衰竭模型可能不是相同的与在能适应的伤寒的乙二酸酯分泌物期间被观察的那些。
Hyperoxaluria (高草酸尿) (CH)单独未同在大肠乙二酸酯(草酸盐)运输上的变化联系在一起，而在净乙二酸酯上的变化(草酸盐运输在末端冒号(末端结肠)从得到分泌物的吸收在模型被观察了与hyperoxalemia (CHH和啊)。血管紧缩素II感受器官(血管紧张素Ⅱ受体)对抗性(拮抗作用)与losartan (氯沙坦) rereduced净大肠乙二酸酯不是分泌物在啊，但CHH。